|1.||Jakubowski, Hieronim: 21 articles (07/2015 - 02/2005)|
|2.||Djuric, Dragan: 6 articles (01/2013 - 01/2009)|
|3.||Stanojlović, Olivera: 6 articles (01/2013 - 05/2009)|
|4.||Jakubowski, H: 6 articles (12/2008 - 02/2000)|
|5.||Hrncić, Dragan: 5 articles (01/2013 - 01/2009)|
|6.||Rasić-Marković, Aleksandra: 5 articles (01/2013 - 01/2009)|
|7.||Djuric, D: 4 articles (06/2015 - 05/2014)|
|8.||Macut, Djuro: 4 articles (01/2012 - 05/2009)|
|9.||Macut, D: 3 articles (06/2014 - 03/2011)|
|10.||Marczak, Lukasz: 3 articles (01/2014 - 10/2010)|
06/01/2014 - "The aim of the study was to examine the effects of chronic exercise training on seizures induced by homocysteine thiolactone (HCT) in adult rats. "
01/01/2013 - "The aim of the present study was to investigate the effects of paradoxical sleep deprivation on homocysteine thiolactone (H)-induced seizures in rats, a model of generalized seizures. "
05/01/2009 - "Two types of seizures in homocysteine thiolactone-treated adult rats, behavioral and electroencephalographic study."
01/01/2014 - "The inactivation of the Pon1 gene in mice causes the accumulation of Hcy-thiolactone in the brain and increases the susceptibility to Hcy-thiolactone-induced seizures. "
01/01/2014 - "The inactivation of the Blmh gene in mice causes accumulation of Hcy-thiolactone in the brain and increases susceptibility to Hcy-thiolactone-induced seizures. "
06/01/2006 - "This article reviews recent studies of Hcy-thiolactone and N-Hcy-protein in the human body, including their roles in autoimmune response, cellular toxicity, and atherosclerosis. "
01/01/2011 - "Recently it has been suggested that a physiological substrate of serum PON1 is homocysteine thiolactone which is a putative risk factor in atherosclerosis. "
01/01/2002 - "Prevention of homocysteine thiolactone induced atherogenesis in rats."
07/07/2000 - "Homocysteine thiolactone and protein homocysteinylation in human endothelial cells: implications for atherosclerosis."
04/01/1999 - "Homocysteine thiolactone is a cyclic thioester that is implicated in the development of atherosclerosis. "
06/01/2015 - "The maximal cardiotoxic effect of homocysteine thiolactone was detected at 1 × 10⁻³ M, which corresponds to severe hyperhomocysteinemia. "
07/01/2012 - "Hyperhomocysteinemia was induced using a reduced form of Hcys (at final dose of 0.1mM) and the most reactive form of Hcys - its cyclic thioester, homocysteine thiolactone (HTL, 1 μM). "
01/01/2011 - "We induced hyperhomocysteinemia using a reduced form of Hcys (final dose, 0.1 mM) and the most reactive form of Hcys, its cyclic thioester, homocysteine thiolactone (HTL, 1 µM). "
12/01/2009 - "Homocysteinylation is a post-translational protein modification which involves homocysteine-thiolactone and may be responsible for many pathophysiological changes secondary to hyperhomocysteinemia. "
10/01/2009 - "Hyperhomocysteinemia induced by feeding rats diets rich in DL-homocysteine thiolactone promotes alterations on carotid reactivity independent of arterial structure."
01/01/1988 - "The findings support the previous demonstration of abnormal homocysteine thiolactone metabolism in arteriosclerosis and cancer."
01/01/1988 - "Homocysteine thiolactone in arteriosclerosis and cancer."
01/01/2003 - "This facile method for quantitation of homocysteine thiolactone opens the way for more detailed clinical studies of its potential role in homocysteine-induced arteriosclerosis and vaso-occlusive disease."
02/01/1989 - "Homocysteinemia and arteriosclerosis: failure to isolate homocysteine thiolactone from plasma and lipoproteins."
05/01/1975 - "The growth disorders associated with homocystinuria, including arteriosclerosis and accelerated growth, are believed to result from increased conversion of methionine to homocysteine thiolactone and homocysteic acid."
|3.||Folic Acid (Vitamin M)
|4.||NG-Nitroarginine Methyl Ester (L-NAME)
|5.||Dizocilpine Maleate (Dizocilpine)
|9.||N-homocysteine thiolactonyl retinamido cobalamin
|10.||N-homocysteine thiolactonyl retinamide