|1.||Sharabi, Yehonatan: 3 articles (03/2015 - 12/2012)|
|2.||Goldstein, David S: 3 articles (03/2015 - 12/2012)|
|3.||Jinsmaa, Yunden: 2 articles (03/2015 - 12/2012)|
|4.||Cooney, Adele: 2 articles (03/2015 - 12/2012)|
|5.||Kopin, Irwin J: 2 articles (12/2014 - 12/2012)|
|6.||Domont, Gilberto B: 1 article (11/2015)|
|7.||Pinheiro, Anderson S: 1 article (11/2015)|
|8.||Eliezer, David: 1 article (11/2015)|
|9.||Follmer, Cristian: 1 article (11/2015)|
|10.||Coelho-Cerqueira, Eduardo: 1 article (11/2015)|
|1.||Parkinson Disease (Parkinson's Disease)
08/01/2012 - "Previous studies have shown it to be covalently modified and potently inhibited by 3,4-dihydroxyphenylacetaldehyde (DOPAL), an endogenous neurotoxin via dopamine catabolism which is relevant to Parkinson's disease. "
11/13/2015 - "Oxidative deamination of dopamine produces the highly toxic aldehyde 3,4-dihydroxyphenylacetaldehyde (DOPAL), enhanced production of which is found in post-mortem brains of Parkinson disease patients. "
12/01/2014 - "According to the "catecholaldehyde hypothesis" for the pathogenesis of Parkinson disease, long-term increased build-up of 3,4-dihydroxyphenylacetaldehyde (DOPAL), the catecholaldehyde metabolite of dopamine, causes or contributes to the eventual death of dopaminergic neurons. "
12/01/2012 - "Vesicular uptake blockade generates the toxic dopamine metabolite 3,4-dihydroxyphenylacetaldehyde in PC12 cells: relevance to the pathogenesis of Parkinson's disease."
01/01/2011 - "[Reactive oxygen species and 3,4-dihydroxyphenylacetaldehyde in pathogenesis of Parkinson disease]."
|4.||Reactive Oxygen Species (Oxygen Radicals)
|5.||L-Lactate Dehydrogenase (Lactate Dehydrogenase)
|6.||Hydrogen Peroxide (Hydroperoxide)