|1.||Beyer, Jochen: 2 articles (03/2009 - 05/2007)|
|2.||Maurer, Hans H: 2 articles (03/2009 - 05/2007)|
|3.||Lindsay, James N: 1 article (11/2009)|
|4.||West, Patrick L: 1 article (11/2009)|
|5.||Horowitz, B Zane: 1 article (11/2009)|
|6.||Montanaro, Marc T: 1 article (11/2009)|
|7.||Boz, Mustafa: 1 article (09/2009)|
|8.||Ilhan, Mustafa: 1 article (09/2009)|
|9.||Arihan, Okan: 1 article (09/2009)|
|10.||Iskit, Alper B: 1 article (09/2009)|
12/31/1996 - "We have previously shown that rats are not susceptible and rabbits only weakly susceptible to coniine-induced arthrogryposis. "
12/31/1996 - "Coniine, an alkaloid from Conium maculatum (poison hemlock), is a known teratogen in many domestic species with maternal ingestion resulting in arthrogryposis of the offspring. "
04/01/1994 - "In summary, the chick embryo provides a reliable and simple experimental animal model of coniine-induced arthrogryposis. "
04/01/1994 - "However, coniine has failed to produce arthrogryposis in rats or mice and is only weakly teratogenic in rabbits. "
07/01/1993 - "Coniine/CM teratogenicity culminates in production of arthrogryposis. "
12/16/1989 - "Rhabdomyolysis and acute tubular necrosis in coniine (hemlock) poisoning."
05/01/2007 - "For diagnosis of such poisonings, multianalyte procedures were developed for detection and validated quantification of the toxic alkaloids aconitine, atropine, colchicine, coniine, cytisine, nicotine and its metabolite cotinine, physostigmine, and scopolamine in plasma using LC-APCI-MS and LC-ESI-MS/MS. After mixed-mode solid-phase extraction of 1 ml of plasma, the analytes were separated using a C8 base select separation column and gradient elution (acetonitrile/ammonium formate, pH 3.5). "
03/10/2009 - "This review describes the toxic alkaloids aconitine, atropine, coniine, colchicine, cytisine, dimethyltryptamine, harmine, harmaline, ibogaine, kawain, mescaline, scopolamine, and taxine, which are often involved in fatal and non-fatal poisonings. "
11/01/2009 - "Conium maculatum is a common weed that causes toxicity by its primary toxin, coniine, which stimulates nicotinic receptors and causes a syndrome of rapidly progressive muscle weakness and paralysis. "
04/01/1990 - "At 25 mg coniine/kg body weight, clinical signs were observed only in quails (2/10) and consisted of excitement, depression, hypermetria, seizures, opisthotonos, and flaccid paralysis. "
|7.||Nicotinic Receptors (Nicotinic Acetylcholine Receptor)