|1.||Wang, Bin: 2 articles (10/2005 - 05/2003)|
|2.||Galeotti, N: 1 article (05/2015)|
|3.||Sanna, M D: 1 article (05/2015)|
|4.||Quattrone, A: 1 article (05/2015)|
|5.||Ghelardini, C: 1 article (05/2015)|
|6.||Peroni, D: 1 article (05/2015)|
|7.||Krieger, Nancy S: 1 article (01/2011)|
|8.||Bushinsky, David A: 1 article (01/2011)|
|9.||Mori, Hidezo: 1 article (10/2006)|
|10.||Ishino, Kozo: 1 article (10/2006)|
10/01/2006 - "At 4h ischemia, the increment in the dialysate NE level (vehicle group, 143+/-30 pg/ml) was suppressed by TMB-8 (25+/-5 pg/ml) but not by DMI (128+/-10 pg/ml) or CTX (122+/-18 pg/ml). "
03/01/1988 - "The results indicate that TMB-8 suppresses the ability of relaxation in non-ischemic myocardium and prevents the impairment of left ventricular relaxation induced by left ventricular global ischemia."
03/01/1988 - "In the ischemic group, the intracoronary infusion of 30 and 100 micrograms/min TMB-8 suppressed the increases of T and left ventricular end-diastolic pressure caused by ischemia and increase of pacing rate. "
03/01/1988 - "The effects of TMB-8, an inhibitor of Ca2+ release from the intracellular store site, on impaired left ventricular relaxation caused by global ischemia were examined in 45 anesthetized dogs. "
03/01/1988 - "[The effects of TMB-8, an intracellular Ca2+ antagonist, on impaired left ventricular relaxation by global ischemia in dog]."
04/01/2000 - "Reduction of neuronal damage in ischemic stroke using a combination therapy of TMB-8 with L-arginine."
04/01/2000 - "These results indicated that a combination of TMB-8 and L-arginine is more effective in treating ischemic stroke by simultaneously reducing calcium-activated proteolysis and improving of cerebral blood flow than using TMB-8 or L-arginine alone."
04/01/2000 - "Treatment with a combination of a calcium antagonist TMB-8 and NO donors, L-arginine and N alpha-benzoyl-L-arginine ethyl ester (BAEE) to prevent experimental ischemic stroke were studied in rats through the permanent occlusion of the middle cerebral artery and common carotid artery for 60 minutes. "
|3.||Asthma (Bronchial Asthma)
08/22/2001 - "Compared to the ATCC strain, 9033 produced high levels of necrosis-associated DNA fragmentation of TMB-8 cells at 4 and 8 h post-treatment. "
04/01/2000 - "Treatment with the combination of TMB-8 and BAEE at time 0, 6 or 24 hrs after ischemia, resulted in the areas of necrosis being reduced by 99%, 96%, and 82%, respectively. "
04/01/2000 - "A combination with TMB-8 and L-arginine at time 0, 6 or 24 hr after ischemia resulted in the areas of necrosis being reduced by 99%, 99%, and 89%, respectively. "
10/01/1995 - "However, when TMB-2, TMB-5 and TMB-8 were administered 6 hr after ischemia, the necrosis areas were reduced less effectively by 83%, 78% and 91%, respectively. "
10/01/1995 - "When the animals were pretreated with TMB-2, TMB-5 and TMB-8, 1 hr before ischemia, the areas of neural necrosis were reduced almost completely by 95%, 99% and 92%, respectively. "
06/01/1990 - "TMB-8 failed to reduce the halothane contracture of MHS muscle but hastened its onset. "
04/01/1987 - "It was found that at the appropriate concentration TMB-8 blocked twitches but neither high K+ nor caffeine induced contractures. "
09/29/1986 - "TMB-8 (5 X 10(-5) - 10(-4) M) blocked electrically evoked twitches but not high K+ (123 mM)- or caffeine (2.36 mM)-induced contractures in isolated, curarized toe muscles. "
09/29/1986 - "TMB-8 can block twitches without blocking high K+ or caffeine induced contractures in frog's skeletal muscle."
05/01/1994 - "Submaximal potassium-induced contractures were markedly depressed while caffeine-induced contractures were only slightly depressed by TMB-8 in the concentration range tested (10(-5)-3 x 10(-4) M). "
|2.||Serotonin (5 Hydroxytryptamine)
|4.||Caffeine (No Doz)
|7.||Ala(2)-MePhe(4)-Gly(5)- Enkephalin (DAGO)
|9.||Intercellular Adhesion Molecule-1 (Intercellular Adhesion Molecule 1)