|1.||Fuss, Ivan J: 2 articles (01/2005 - 01/2002)|
|2.||Strober, Warren: 2 articles (01/2005 - 01/2002)|
|3.||Boirivant, Monica: 2 articles (01/2005 - 01/2002)|
|4.||Zhou, You: 1 article (11/2015)|
|5.||Su, Qiang: 1 article (11/2015)|
|6.||Liu, Yangchun: 1 article (11/2015)|
|7.||Li, Lang: 1 article (11/2015)|
|8.||Wang, Jiangyou: 1 article (11/2015)|
|9.||Firouzi, Masoumeh: 1 article (04/2015)|
|10.||Zadegan, Shayan Abdollah: 1 article (04/2015)|
05/01/1985 - "[Histopathological diagnosis of early stages of myocardial infarction--applications of the improved hematoxylin basic fuchsin picric acid (HBFP) staining method to human autopsy hearts]."
06/01/1983 - "Comparison of the haematoxylin basic fuchsin picric acid method and the fluorescence of haematoxylin and eosin stained sections for the identification of early myocardial infarction."
01/01/1982 - "[Reproducibility, evidence and autolysis resistance of Lie's hematoxylin-based fuchsin-picric acid staining for determining fresh myocardial infarctions]."
02/01/1989 - "The immunostaining patterns were compared with the appearances seen in adjacent sections stained by the routine haematoxylin and eosin (H & E) and phosphotungstic acid haematoxylin (PTAH) methods and a method previously recommended for the detection of early myocardial infarction, the haematoxylin basic fuchsin picric acid (HBFP) stain. "
09/01/1984 - "[Comparison of acid fuchsin (AF) staining and hematoxylin-basic fuchsin-picric acid (HBFP) staining for demonstration of early myocardial infarction]."
09/01/1981 - "Paraffin sections of myocardium stained by von Kóssa's method or by haematoxylin-basic fuchsin-picric acid improved the detection of myocardial necrosis. "
01/01/1978 - "Autopsy showed widespread intravascular sickling; staining with hematoxylin-basic fuchsin-picric acid demonstrated newly developed massive myocardial necrosis, a rarely documented finding. "
07/01/1975 - "[Validity of hematoxylin-basic fuchsin-picric acid (hbfp) in detection of early myocardial necrosis in the mice]."
|4.||Myocardial Ischemia (Ischemic Heart Diseases)
08/25/2004 - "Under a light microscope, the pathological examination revealed that all the cells were swollen and in red color when the cardiac section was stained with hematoxylin basic fuchsin picric acid (HBFP), which indicated a typical change in the myocardial ischemia. "
05/01/1985 - "Hematoxylin basic fuchsin picric acid (HBFP) staining method has been controversial on reliability for the morphologic diagnosis of early stages of myocardial ischemia. "
01/01/1978 - "The effect of this treatment was evaluated histologically in studies on acute ischemia by the use of the hematoxylin-basic fuchsin-picric acid stain for early myocardial ischemia. "
05/01/1974 - "[Histochemical technic of Hematoxylin-Basic Fuchsin-Picric acid (HBFP) in the study of sudden death due to myocardial ischemia]."
01/01/2011 - "The myocardial injury was evaluated quantitatively in terms of the serum cardiac troponin I (cTnI) levels, the myocardial ischemia/hypoxia area, and the integral optical density (IOD) of haematoxylin-basic fuchsin-picric acid (HBFP) staining, and qualitatively in terms of the myocardial ultrastructure. "
04/15/1999 - "We previously demonstrated that 2,4,6-trinitrophenol (TNP)-OVA immunization leads to a transmural colitis in the IL-2-/- mouse that is caused by IL-12-driven CD4+ Th1 T cells and resembles human Crohn's disease. "
10/01/1997 - "A severe, Th1-mediated experimental colitis with similarities to inflammatory bowel disease in humans can be induced by a single injection of 2,4,6-trinitrophenol (TNP)-substituted protein plus adjuvant in IL-2-/- mice. "
01/01/2005 - "In previous studies, we have shown that the oral administration of colonic proteins that have been haptenated (i.e., haptenated colonic proteins [HCPs]) with trinitrophenol (TNP) can protect mice from the subsequent induction of trinitrobenzene sulfonic acid colitis. "
01/15/2002 - "In initial studies, we showed that the feeding of trinitrophenol-haptenated colonic protein to SJL/J mice induces CD4(+) regulatory T cells that transfer protection from induction of TNBS-colitis, and that such protection correlates with cells producing TGF-beta, not IL-10. "
01/01/1997 - "Gene-targeted mice lacking the IL-2 gene (IL-2 -/- mice) develop various forms of autoimmunity as well as severe colitis, either spontaneously in a conventional environment or after immunization with 2,4,6-trinitrophenol (TNP)-conjugated keyhole limpet hemocyanin (KLH) in a specific pathogen-free environment. "
|6.||Proteins (Proteins, Gene)
|7.||Immunoglobulin E (IgE)
|9.||Interleukin-10 (Interleukin 10)
|10.||Transforming Growth Factor beta (TGF-beta)