|1.||Phillis, J W: 6 articles (09/2003 - 01/2000)|
|2.||O'Regan, M H: 6 articles (09/2003 - 01/2000)|
|3.||Voelker, Dennis R: 4 articles (01/2015 - 04/2005)|
|4.||Choi, Jae-Yeon: 4 articles (01/2015 - 04/2005)|
|5.||Maria, Durvanei Augusto: 4 articles (07/2013 - 01/2012)|
|6.||Ferreira, Adilson Kleber: 4 articles (07/2013 - 01/2012)|
|7.||Chierice, Gilberto Orivaldo: 4 articles (07/2013 - 01/2012)|
|8.||Morvan, Daniel: 4 articles (03/2007 - 03/2002)|
|9.||Heerschap, Arend: 3 articles (09/2014 - 12/2012)|
|10.||Klomp, Dennis W J: 3 articles (01/2014 - 12/2011)|
09/01/2014 - "(31)P-MR spectra from the IDH1-mutant tumor displayed a pattern distinct from that of the three IDH1 wild-type tumors, characterized by decreased levels of phosphoethanolamine and increased levels of glycerophosphocholine. "
01/01/2013 - "In a recent report, we showed that synthetic phosphoethanolamine (Pho-s) has a broad antitumor activity on a variety of tumor cells and showed potent inhibitor effects on tumor progress in vivo. "
02/01/2009 - "Phosphoethanolamine was increased in cancer compared with normal tissue (P = 0.0001). "
12/20/1993 - "Many human tumors contain high concentrations of ethanolamine phosphate (EtnP). "
11/01/1979 - "A possible role of phosphoethanolamine in the growth of mammary tumor cells as well as of normal mammary epithelial cells and other tissues is discussed."
|2.||Parkinson Disease (Parkinson's Disease)
07/01/1986 - "We measured the concentration of gamma-aminobutyric acid (GABA), glutamic acid, and o-phosphoethanolamine in autopsied brain of 9 patients who died with idiopathic Parkinson's disease and 10 control subjects. "
01/01/1988 - "Results showed statistically significant decreases in the levels of gamma-aminobutyric acid, homocarnosine, phosphoethanolamine, and threonine, and elevation of ornithine levels, in the CSF of de novo patients with Parkinson's disease compared with controls. "
01/01/2001 - "Nigral activity of the catabolic enzyme phospholipase A2 was normal in the Parkinson's disease group, whereas that of the biosynthetic enzymes phosphoethanolamine cytidylyltransferase, phosphocholine cytidylyltransferase, and phosphatidylserine synthase were elevated 193, 48 and 38%, respectively, possibly representing a compensatory response to repair membrane phospholipids. "
12/01/1959 - "A genetical study of ethanolamine phosphate excretion in hypophosphatasia."
12/01/2012 - "Phosphoethanolamine normal range in pediatric urines for hypophosphatasia screening."
01/01/1994 - "Urinary phosphoethanolamine, a typical marker of hypophosphatasia, was absent from all specimens, which rules out the possible diagnosis of such disorder in these patients. "
06/01/1980 - "[Hypophosphatasia with normal urinary phosphoethanolamine in a 22-month-old girl]."
01/01/1979 - "Increased urinary excretion of phosphorylethanolamine (P.E.A.) is one of the salient features of hypophosphatasia. "
|4.||Brain Ischemia (Cerebral Ischemia)
11/01/1998 - "The report concerns mechanisms for the increase of extracellular levels of ethanolamine and phosphoethanolamine in CNS regions, such as the hippocampus, in transient brain ischemia, hypoglycemia, seizures, etc. L-Serine (2.5-10 mM), D-serine (10 mM), or ethanolamine (10 mM) was administered for 20 min via a microdialysis tubing to the hippocampus of unanesthetized rabbits. "
01/01/2001 - "Superfusate aspartate, glutamate, phosphoethanolamine, taurine, and GABA were significantly elevated by cerebral ischemia, then declined during reperfusion. "
08/07/1999 - "Cerebral ischemia elicited increased superfusate levels of aspartate, glutamate, phosphoethanolamine, taurine, gamma-aminobutyric acid (GABA) and arachidonic acid when compared with basal levels. "
05/30/1997 - "Twenty minutes of four vessel cerebral ischemia caused significant increases in cortical superfusate levels of aspartate, glutamate, GABA, taurine and phosphoethanolamine. "
05/21/1986 - "During brain ischemia, a principal pathway for degradation of ethanolamine glycerophospholipids may be by reversal of phosphoethanolamine transferase followed by hydrolysis of diacylglycerols by the lipase."
|5.||Breast Neoplasms (Breast Cancer)
07/01/2013 - "Synthetic phosphoethanolamine induces cell cycle arrest and apoptosis in human breast cancer MCF-7 cells through the mitochondrial pathway."
08/01/2012 - "In addition, data of three patients with breast cancer showed higher levels of phosphocholine and phosphoethanolamine compared with healthy volunteers. "
10/01/2008 - "Metabolic labeling with [14C]ethanolamine revealed a reduced ECT activity in MCF-7 breast cancer cells, which led to an accumulation of phosphoethanolamine and a decrease in PE synthesis in comparison with MCF-10A mammary epithelial cells. "
07/01/1991 - "Analysis of cytosolic phosphoethanolamine and ethanolamine and their correlation with prognostic factors in breast cancer."
10/01/2013 - "The T2 values of phosphomonoesters (PME) and PDE in breast cancer tissue (ductulolobular carcinoma) ranged between 170 and 210 ms, and the PME to PDE ratios were calculated to be phosphoethanolamine/glycerophosphoethanolamine = 2.7, phosphocholine/glycerophosphocholine = 1.8 and PME/PDE = 2.3. Considering the relatively short T2 values of the metabolites in breast tissue at 7 T, the echo spacing can be short without compromising spectral resolution, whilst maximizing the sensitivity."
|1.||gamma-Aminobutyric Acid (GABA)
|2.||Polyethylene Glycols (Polyethylene Glycol)
|3.||Small Interfering RNA (siRNA)
|8.||Glutamic Acid (Glutamate)
|9.||Aspartic Acid (Aspartate)
|10.||Choline (Choline Chloride)
|2.||Heterologous Transplantation (Xenotransplantation)
|3.||Drug Therapy (Chemotherapy)