|1.||Yurdakan, Gamze: 4 articles (05/2014 - 02/2012)|
|2.||Yilmaz-Sipahi, Emine: 3 articles (05/2014 - 02/2012)|
|3.||Sipahi, Emine Yilmaz: 3 articles (11/2012 - 12/2004)|
|4.||Niyazi Ayoglu, Ferruh: 2 articles (03/2005 - 03/2002)|
|5.||Barut, Figen: 2 articles (03/2005 - 12/2004)|
|6.||Comert, Mustafa: 2 articles (03/2005 - 12/2004)|
|7.||Ustun, Huseyin: 2 articles (03/2005 - 12/2004)|
|8.||Sipahi, Tunc Hakan: 2 articles (03/2005 - 12/2004)|
|9.||Delclaux, Christophe: 2 articles (05/2003 - 09/2002)|
|10.||Azoulay, Elie: 2 articles (05/2003 - 09/2002)|
11/01/1982 - "[Immuno-electron microscopical study on pulmonary edema induced by alpha-naphthylthiourea]."
11/01/1981 - "[Pulmonary edema induced by alpha-naphthylthiourea--usual ultrastructural, immuno-electron microscopical and freeze fracture studies]."
02/21/2000 - "ANTU(H) alone resulted in an increase in pulmonary edema leading to animal death. "
06/01/1996 - "The alpha-naphthylthiourea produced an acute permeability pulmonary edema detected by lung leak over the 180-min ex vivo period of monitoring the isolated perfused lung (leak = 8+/-mL; wet/dry weight ratio 14.7+/-2; lavage protein 3.1+/-1 mg/mL). "
06/01/1996 - "Acute permeability pulmonary edema was induced with an injection of alpha-naphthylthiourea, and lung leak was assessed in an isolated perfused lung model over 180 mins. "
06/01/1984 - "However, platelet depletion studies suggest that 15-M-PGE1 has additional effects, possibly on endothelial cells and/or vascular smooth muscle cells that contribute to the potentiation of ANTU -induced lung injury."
06/01/1984 - "These studies indicate that systemic treatment of rats with 15-M-PGE1 will potentiate ANTU -induced lung injury. "
02/01/1995 - "ANTU alone and HCl alone caused a moderate histologic lung injury, increased wet/dry weight ratio, and resulted in a small increase in flux. "
04/01/1986 - "Transvascular clearance and distribution of charged macromolecules in ANTU lung injury."
06/01/1984 - "Platelet depletion, but not neutrophil depletion, also potentiated ANTU -induced lung injury, suggesting a protective role for platelets. "
08/15/2001 - "In keeping with this explanation, the lower Vuip and Crs were before HV, the more severe HV-induced edema was in alpha-naphthylthiourea-injected rats. "
08/15/2001 - "Changes in Vuip and in Crs produced by HV were correlated with edema severity in normal rats or rats with lungs preinjured with alpha-naphthylthiourea. "
05/01/1995 - "ANTU alone produced moderate permeability edema with significant increases in extravascular lung water (Qwl), dry lung weight (DLW), and albumin distribution space in lungs (ASp). "
02/01/1987 - "Sequential changes in lung metabolism, permeability, and edema after ANTU."
07/01/1985 - "Hydrostatic edema (HE) was induced by raising the venous pressure, and permeability edema (PE) was induced with alpha-naphthylthiourea. "
|4.||Acute Lung Injury
11/01/2012 - "Dexmedetomidine treatment has demonstrated a potential benefit by preventing ANTU-induced acute lung injury in an experimental rat model. "
11/01/2012 - "We assessed the effects of dexmedetomidine in a rat model of α-naphthylthiourea (ANTU)-induced acute lung injury. "
09/01/2002 - "The effects of granulocyte colony-stimulating factor given alone or after alpha-naphthylthiourea used to induce acute lung injury were investigated. "
02/01/1995 - "The effect of G-CSF pretreatment on experimental acute lung injury was studied in Sprague-Dawley rats receiving one of the following treatments: (1) G-CSF 50 micrograms/kg subcutaneously twice daily beginning 2 d prior to being killed; (2) ANTU 50 mg/kg intraperitoneally; (3) ANTU+G-CSF 50, 25, or 12.5 micrograms/kg; (4) HCl 0.6 ml of a 0.1 N solution intratracheally; (5) HCl+G-CSF 50 or 25 micrograms/kg; (6) control solutions. "
01/01/1987 - "We conclude that lungs are capable of hydrolysis of triglyceride contained in chylomicrons and that this endothelial cell associated metabolic function is not altered by acute lung injury caused by ANTU."
06/01/1978 - "In some experimental models, such as alloxan, leakage is due to irreversible injury to the alveolar wall; in other models, including ANTU, oedema formation has been shown to depend upon minor and reversible changes in pulmonary vascular endothelium similar to those that cause exudate formation in areas of acute inflammation. "
11/01/2012 - "The inflammation, hemorrhage, and edema scores in the ANTU group were significantly greater than those in the control or dexmedetomidine 50-μg/kg group (P < 0.01). "
|1.||alpha-naphthyl thiourea (ANTU)
|3.||Endothelin Receptors (Endothelin Receptor)
|6.||Oleic Acid (Oleate)
|7.||tranilast (N 5')
|5.||Artificial Respiration (Mechanical Ventilation)