|1.||Efimov, Igor R: 2 articles (01/2012 - 01/2004)|
|2.||Rassier, Dilson E: 2 articles (11/2008 - 03/2005)|
|3.||Jin, Zhe Hu: 1 article (09/2015)|
|4.||Jang, Ji Hyun: 1 article (09/2015)|
|5.||Zhao, Zai Hao: 1 article (09/2015)|
|6.||Shin, Dong Hoon: 1 article (09/2015)|
|7.||Kim, Sung Joon: 1 article (09/2015)|
|8.||Wang, Yue: 1 article (09/2015)|
|9.||Jin, Chun Zi: 1 article (09/2015)|
|10.||Youm, Jae Boum: 1 article (09/2015)|
09/03/2010 - "In 18 muscle strips contractures were prevented by means of butanedione monoxime (BDM). "
01/01/2007 - "Finally, delayed contracture with A(1)AR agonism/overexpression or ischaemic 2,3-butanedione monoxime (BDM; 5 microm to target Ca(2+) cross-bridge formation) was linked to enhanced postischaemic outcomes. "
09/01/1997 - "Limbs in the University of Wisconsin solution plus butanedione monoxime group had more flexible ankle joints and pliable muscle (i.e., less contracture) than those in the University of Wisconsin solution and no flushout groups. "
07/01/1994 - "Mechanism of action of 2,3-butanedione monoxime on contracture during metabolic inhibition."
09/01/1991 - "Effects of 2,3-butanedione monoxime (BDM) on contracture and injury of isolated rat myocytes following metabolic inhibition and ischemia."
01/01/1999 - "The efficacy of 2,3-butanedione monoxime (BDM) as additive to St. Thomas Hospital II solution (STH) as compared to initial BDM reperfusion with regard to myocardial ischaemia/reperfusion injury was investigated in isolated guinea pig hearts. "
01/01/1996 - "Contraction uncoupling by a negative inotropic and vasodilating agent such as 2,3-butanedione monoxime (BDM) may be superior to warm cardioplegic reperfusion in reducing reperfusion damage. "
01/01/1998 - "The effect of 20 mmol/L butanedione monoxime on myocardial ischemia/reperfusion damage was studied in isolated guinea pig hearts. "
01/01/1998 - "Optimizing the oxygen balance during initial reperfusion with 2,3-butanedione monoxime attenuates cardiac reperfusion injury."
11/25/1997 - "In contrast, rat cardiomyocytes cultured under very similar and optimal conditions exhibit very different characteristics during their adaptation in long-term cultures: (1) although 85-90% of freshly isolated cells are also rod-shaped and Ca2+ tolerant they exhibit slow spontaneous contractions; (2) they round up during the first few days, and during the first week they dedifferentiate, losing their regular striated appearance; (3) they spread, becoming irregularly shaped, and, unlike the guinea pig cardiomyocytes, they do not form confluent layers, no matter what the plating density is; (4) they atrophy at a very early stage in the cultures, so that by the fourth week, they have lost most of their myofibrils; (5) the initial rounding up is largely eliminated by exposure for 24 h to 1 microM ryanodine or 20 mM butanedione monoxime, compounds that suppress the spontaneous contractions. "
11/01/1996 - "To determine whether myofibrillar atrophy results indirectly from loss of mechanical signals or directly from alterations in intracellular Ca2+ concentration ([Ca2+]i), contractile activity was inhibited with verapamil (10 microM) or 2,3-butanedione monoxime (BDM), and their effects on cell shortening, [Ca2+]i, and myosin heavy chain (MHC) turnover were assessed. "
03/01/1993 - "This study shows that relatively low concentrations of 2,3-butanedione monoxime, given before global ischemia and early during reperfusion of isolated hearts, can protect against dysrhythmias and improve return of myocardial and vascular function."
07/01/2004 - "Isolated and perfused rat hearts were subjected to 30 min of global ischemia, followed by reperfusion with or without the contractile blocker 2,3-butanedione monoxime (BDM). "
11/01/1993 - "The influence of 2,3-butanedione monoxime (BDM) on function and subcellular energy status in isolated perfused guinea pig hearts was examined during ischemia and reperfusion. "
03/01/1993 - "Hearts were divided into four groups (n = 10 each) treated with 0, 3, 5, or 10 mmol/L of 2,3-butanedione monoxime added to the perfusate for 10 minutes before and during ischemia and for the first 10 minutes of reperfusion. "
03/01/1993 - "Effects of 2,3-butanedione monoxime in isolated hearts: protection during reperfusion after global ischemia."
06/27/1969 - "[Study of the applicability of diacetylmonoxime in humans as an antidote in poisoning with the organophosphate sarin]."
08/01/1959 - "Mechanism of the prophylactic action of diacetylmonoxime against sarin poisoning."
11/07/1958 - "Pyridine-2-aldoxime methiodide and diacetyl monoxime against organophosphorus poisoning."
08/01/1959 - "Prophylactic and therapeutic effects of pyridine-2-aldoxime methiodide and diacetyl monoxime against poisoning by organophosphorus compounds."
09/01/1957 - "Three oximes, monoisonitrosoacetone (MINA), pyridine-2-aldoxime methiodide (PAM) and diacetylmonoxime (DAM), have been examined in combination with atropine as antidotes in sarin poisoning. "
|8.||Adenosine Triphosphate (ATP)
|9.||pralidoxime (Protopam Chloride)
|2.||Cardiopulmonary Resuscitation (CPR)
|3.||Induced Heart Arrest (Cardioplegia)