|1.||Cantor, Glenn H: 2 articles (02/2011 - 12/2010)|
|2.||Coen, Muireann: 2 articles (02/2011 - 08/2009)|
|3.||Nicholson, Jeremy K: 2 articles (02/2011 - 08/2009)|
|4.||Lindon, John C: 2 articles (02/2011 - 08/2009)|
|5.||Ohno, Yasuo: 1 article (01/2013)|
|6.||Maruyama, Toshiyuki: 1 article (01/2013)|
|7.||Yamada, Hiroshi: 1 article (01/2013)|
|8.||Ono, Atsushi: 1 article (01/2013)|
|9.||Yamate, Jyoji: 1 article (01/2013)|
|10.||Hanafusa, Hiroyuki: 1 article (01/2013)|
01/01/1984 - "Papillary necrosis was induced in rats by intravenous administration of bromoethyleneamine hydrobromide (BEA) 6 weeks prior to the study, in order to assess the role of deep nephrons in this process. "
02/01/2011 - "2-Bromoethanamine (BEA) causes renal papillary necrosis (RPN) in rats after a single dose and has been widely used as a model compound for studying the lesion. "
12/01/2010 - "Effects of 2-bromoethanamine on TonEBP expression and its possible role in induction of renal papillary necrosis in mice."
01/01/1995 - "Renal papillary necrosis (RPN) was induced in Fischer 344 (F344) rats (n = 4) using 2-bromoethanamine hydrobromide (BEA) dosed at 150 mg/kg, and in multimammate desert mice (Mastomys natalensis) at 150 and 250 mg/kg (n = 4 per group). "
01/01/1995 - "We have assessed the importance of inner medullary structures for the effects of CNS stimulation by examining its ability to alter renal excretion in rats with papillary necrosis, induced 2 d earlier with 2-bromoethylamine hydrobromide (BEA), 250 mg kg-1 body wt i.v. Male Lewis x DA rats were divided into a BEA-treated group (n = 6) and a control group receiving vehicle alone (n = 6). "
|2.||Body Weight (Weight, Body)
01/01/1992 - "Sequential light microscopic and ultrastructural examination of kidneys from male and light microscopic examination of female Mongolian gerbils given 250 mg 2-bromoethylamine hydrobromide (BEA)/kg body weight ip were performed. "
08/01/1991 - "Female Swiss ICR mice were injected ip with 100 or 300 mg 2-bromoethylamine hydrobromide (BEA)/kg body weight. "
12/01/1988 - "In order to examine this hypothesis normotensive and spontaneously hypertensive rats of the Münster strain (SHR) were chemically renomedullectomized by 2-bromoethylamine hydrobromide (BEA), injected intraperitoneally in a single dose of 200 mg/kg body weight. "
10/01/1982 - "Renal function was investigated in rats 3 d or 4 wk after an injection of 2-bromoethylamine hydrobromide (BEA) 40-125 mg/kg body weight. "
08/01/1989 - "1. Chemical renal medullectomy was performed in Wistar rats by intraperitoneal injection of 2-bromoethylamine hydrobromide (200 mg/kg body weight). "
|3.||Hypertension (High Blood Pressure)
04/01/1988 - "The features of hypertension produced in the rat by chemical medullectomy with 2-bromoethylamine hydrobromide are described. "
08/01/1985 - "We have investigated the mechanisms by which chemical renal medullectomy with 2-bromoethylamine hydrobromide (200 mg/kg body wt) produces hypertension in rats. "
05/01/1987 - "Experimental renal papillary necrosis induced by 2-bromoethylamine hydrobromide (BEA) results in hypertension in the rat. "
12/01/1987 - "While pretreatment with aprotinin or indomethacin failed to inhibit the depressor action, 2-bromoethylamine hydrobromide (BEA), which is known to induce necrosis of the renal papilla, produced complete abolition of the depressor effect of an intrarenal injection of MK-422 in NE-induced hypertension. "
08/01/1986 - "The induction of selective renal medullary damage by 2-bromoethylamine hydrobromide (BEA) results in polyuria and raised blood pressure. "
11/01/1984 - "The rat renal papilla was selectively destroyed by 2-bromoethylamine hydrobromide; increasing doses produced a graded severity of histological damage, polyuria and a reduction in urinary prostaglandin E2 excretion. "
|5.||Renal Insufficiency (Renal Failure)
04/01/1991 - "Different models of site specific experimental renal failure (ERF) have been developed in the rat; proximal tubular necrosis, induced by cisplatin; papillary necrosis, induced by 2-bromoethylamine, and glomerulonephritis, induced by sodium aurothiomalate or by antiglomerular basement membrane antibody. "
|2.||atrial natriuretic factor prohormone (103-126)
|3.||Glutathione (Reduced Glutathione)