|1.||Cuzzocrea, Salvatore: 60 articles (09/2015 - 05/2002)|
|2.||Mazzon, Emanuela: 53 articles (06/2015 - 05/2002)|
|3.||Di Paola, Rosanna: 41 articles (01/2014 - 08/2002)|
|4.||Genovese, Tiziana: 37 articles (10/2011 - 12/2003)|
|5.||Bramanti, Placido: 31 articles (06/2015 - 04/2005)|
|6.||Esposito, Emanuela: 27 articles (09/2015 - 07/2006)|
|7.||Muià, Carmelo: 21 articles (08/2008 - 09/2004)|
|8.||Crisafulli, Concetta: 17 articles (02/2010 - 01/2006)|
|9.||Paterniti, Irene: 11 articles (01/2014 - 05/2009)|
|10.||Galuppo, Maria: 10 articles (06/2015 - 05/2009)|
08/03/2001 - "When compared to nonpreconditioned controls, preceding preconditioning improved postischemic cardiac performance and significantly decreased test ischemia/reperfusion-induced formation of free nitrotyrosine measured in the perfusate as a marker for cardiac endogenous ONOO(-) formation. "
07/01/2003 - "The purpose of this study is to evaluate the difference in nitrotyrosine formation and infarct volume between permanent and transient focal ischemia in rats. "
04/01/2015 - "Surprisingly, no positive correlation was found between increased nitrotyrosine levels and mitochondrial fission, particularly in ischemia resistant CA3 and dentate gyrus neurons. "
04/01/2013 - "Paralleling the ischemia-induced nuclear import of Keap1, increased nitrotyrosine immunoreactivity in endothelial cells was also observed. "
01/01/2012 - "In other leukocytes, nitrotyrosine generation peaked at 1 hour of ischemia and again at 18 hours of reperfusion. "
|2.||Hypertension (High Blood Pressure)
01/01/2008 - "HHQ inhibited the CQA-induced improvement in hypertension, urinary NO metabolites or H(2)O(2) excretion, endothelial dysfunction, and nitrotyrosine deposits in aortas in SHR. "
10/04/2013 - "Along with the development of hypertension in SHR rats, VPO1 expression was up-regulated together with a significant increase in NOX activity, HOCl production, 3-nitrotyrosine content, and plasma H2O2 level compared with WKYs at 8, 13 and 20 weeks of age. "
11/01/2005 - "Oxidative stress was determined by quantitative immunoblotting for nitrotyrosine, and T-cell and macrophage content by immunostaining, in offspring kidneys before and after the onset of hypertension. "
04/01/2005 - "Coupled to significant increases in nitrotyrosine levels in the kidney, this suggests a pathophysiological role of this protein in hypertension and oxidative stress. "
08/01/2002 - "Hypertension was present in the HFS group at 2 years as was a significant accumulation, in various tissues, of nitrotyrosine, which is the footprint of NO inactivation by ROS. "
07/15/1996 - "pylori infection resulted in a significant reduction in iNOS and nitrotyrosine staining and a marginally significant reduction in apoptosis. "
10/01/2001 - "The present studies investigated the role of inflammatory cells in nitrotyrosine formation during mycoplasmal infection. "
04/01/2014 - "We conclude that significantly lower levels of 3-nitrotyrosine in pregnant women with CMV infection may indicate an increase in the antioxidant defence mechanisms in these patients."
03/01/2013 - "bovis infection interaction was observed for any parameter measured, with the exception of nitrotyrosine immunostaining in the cecum. "
12/01/2005 - "Protein 3-nitrotyrosine formation during Trypanosoma cruzi infection in mice."
04/01/2013 - "Significant correlations between change in MAGE, change in IMT and change in fasting and interprandial inflammation score and nitrotyrosine plasma levels were found. "
01/01/2012 - "This effect was accompanied by diminished inflammation, as indicated by reduced microgliosis (-20%, P=0.002) and down-regulation of other proinflammatory mediators, and resulted in less oxidative stress, as nitrotyrosine levels declined (-28%, P=0.029). "
04/01/2011 - "The irreversible inflammation induced an increase in the signal intensities for nitrotyrosine and iNOS and a decrease for the α(1)-, β(1)-, and α(2)-subunits of sGC in odontoblasts. "
11/01/2010 - "Moreover, measurements of inflammation/monocyte/macrophage (Mo/MФ) burden, including CD68, ITGAM, EMR-1 and nitrotyrosine were reduced in hSTAT3-HCD-treated animals, while foxp3 (Tregs) and SOCS1 expression were increased. "
07/01/2010 - "The insulitis score and nitrotyrosine staining were reduced, whereas the pancreatic islets and the beta-cell area were increased significantly in the treated group, indicating the reduction of inflammation and nitrosative stress and an early regeneration of beta-cell mass in the pancreas. "
01/30/2006 - "In contrast, the degree of: (a) spinal cord inflammation and tissue injury (histological score), (b) nitrotyrosine, (c) PARS, and (d) neutrophils infiltration was markedly reduced in spinal cord tissue obtained from young rats. "
01/01/2014 - "Treatment with DHA significantly reduced: (1) the degree of spinal cord inflammation and tissue injury, (2) pro-inflammatory cytokine expression (TNF-α), (3) nitrotyrosine formation, (4) glial fibrillary acidic protein (GFAP) expression, and (5) apoptosis (Fas-L, Bax, and Bcl-2 expression). "
08/01/2009 - "The degree of spinal cord inflammation, nitrotyrosine, poli (ADP-ribosio) synthetase (PARS) and neutrophilic infiltration was markedly reduced. "
03/01/2007 - "Treatment of the mice with z-VAD-fmk, a potent broad specific caspase inhibitor, significantly reduced the degree of (1) spinal cord inflammation and tissue injury (histological score), (2) neutrophil infiltration (myeloperoxidase activity), (3) nitrotyrosine formation, and (4) apoptosis (TUNEL staining and Bax and Bcl-2 expression). "
06/01/2006 - "The degree of (a) spinal cord inflammation and tissue injury (histological score), (b) nitrotyrosine, (c) poly(adenosine diphosphate-ribose), (d) neutrophils infiltration, and (e) the activation of signal transducer and activator transcription 3 was markedly reduced in spinal cord tissue obtained from H. "
|1.||NADPH Oxidase (NAD(P)H oxidase)
|3.||Nitric Oxide Synthase Type II (Inducible Nitric Oxide Synthase)
|4.||Nitric Oxide Synthase (NO Synthase)
|8.||Nitric Oxide (Nitrogen Monoxide)
|9.||Peroxisome Proliferator-Activated Receptors (PPAR)
|2.||Drug Therapy (Chemotherapy)
|4.||Induced Heart Arrest (Cardioplegia)
|5.||Homologous Transplantation (Allograft)