|1.||Matsui, Sei Ichi: 1 article (08/2005)|
|2.||Beerman, Terry A: 1 article (08/2005)|
|3.||Tu, Lan Chun: 1 article (08/2005)|
|4.||Beerman, Terry: 1 article (03/2005)|
|5.||Liang, Ping: 1 article (03/2005)|
|6.||Li, Fengzhi: 1 article (03/2005)|
|7.||Altieri, Dario C: 1 article (03/2005)|
|8.||Song, Lei: 1 article (03/2005)|
|9.||Pan, Dalin: 1 article (03/2005)|
|10.||Ling, Xiang: 1 article (03/2005)|
|1.||Chromosome Aberrations (Chromosome Abnormalities)
08/01/2005 - "This study examined the potential of the monofunctional DNA alkylating agent hedamycin, a powerful inhibitor of DNA replication, to induce DNA strand breaks, phosphorylated H2AX (gammaH2AX) foci, and chromosome aberrations. "
08/01/2005 - "Hedamycin, a DNA alkylator, induces (gamma)H2AX and chromosome aberrations: involvement of phosphatidylinositol 3-kinase-related kinases and DNA replication fork movement."
|4.||Ataxia Telangiectasia (Louis Bar Syndrome)
08/01/2005 - "Similarly, hedamycin induction of gammaH2AX is not dependent on ataxia telangiectasia mutated or DNA-protein kinase, and pretreatment of cells with the phosphatidylinositol 3-kinase-related kinase inhibitor caffeine did not substantially reduce induction of H2AX phosphorylation by hedamycin. "
|1.||DNA (Deoxyribonucleic Acid)
|2.||A-Form DNA (A-DNA)
|3.||Protein Kinases (Protein Kinase)
|6.||Caffeine (No Doz)