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D J MacEwan Selected Research

Necrosis

12/2004Granulocyte macrophage-colony stimulating factor and interleukin-3 increase expression of type II tumour necrosis factor receptor, increasing susceptibility to tumour necrosis factor-induced apoptosis. Control of leukaemia cell life/death switching.
11/2001Type II tumour necrosis factor-alpha receptor (TNFR2) activates c-Jun N-terminal kinase (JNK) but not mitogen-activated protein kinase (MAPK) or p38 MAPK pathways.
09/2001Modulated kinase activities in cells undergoing tumour necrosis factor-induced apoptotic cell death.
03/2001Stimulation of stress-activated but not mitogen-activated protein kinases by tumour necrosis factor receptor subtypes in airway smooth muscle.
08/2000Unmodified calcium concentrations in tumour necrosis factor receptor subtype-mediated apoptotic cell death.
03/2000Selective down-regulation of the G(q)alpha/G11alpha G-protein family in tumour necrosis factor-alpha induced cell death.

Articles by Research Topic

Disease

6Necrosis
12/2004 - 03/2000

Drug/Important Bio-Agent (IBA)

1Granulocyte-Macrophage Colony-Stimulating Factor (GM-CSF)IBA
12/2004
1Interleukin-3 (Interleukin 3)IBA
12/2004
1p38 Mitogen-Activated Protein KinasesIBA
11/2001
1JNK Mitogen-Activated Protein KinasesIBA
11/2001
1Protein Kinases (Protein Kinase)IBA
11/2001
1Type II Tumor Necrosis Factor ReceptorsIBA
11/2001
1Phosphotransferases (Kinase)IBA
09/2001
1Mitogen-Activated Protein KinasesIBA
03/2001
1CalciumIBA
08/2000
1GTP-Binding Proteins (G-Protein)IBA
03/2000