PRODUCTS
SERVICES
COMPANY
CONTACT
FAQ
Search
Dictionary
Mobile
Login
Login/Nickname:
Password:
Remember login
Login
Send password reminder...
Ya-Ching Hsieh Selected Research
Estrogens (Estrogen)
10/2008
Mechanism of estrogen-mediated improvement in cardiac function after trauma-hemorrhage: p38-dependent normalization of cardiac Akt phosphorylation and glycogen levels.
04/2008
Mitogen activated protein kinase (MAPK) mediates non-genomic pathway of estrogen on T cell cytokine production following trauma-hemorrhage.
03/2008
Estrogen-mediated activation of non-genomic pathway improves macrophages cytokine production following trauma-hemorrhage.
09/2007
Mechanism of the nongenomic effects of estrogen on intestinal myeloperoxidase activity following trauma-hemorrhage: up-regulation of the PI-3K/Akt pathway.
06/2007
The role of estrogen receptor subtypes in ameliorating hepatic injury following trauma-hemorrhage.
06/2007
Role of p38 mitogen-activated protein kinase pathway in estrogen-mediated cardioprotection following trauma-hemorrhage.
06/2007
The PI3K/Akt pathway mediates the nongenomic cardioprotective effects of estrogen following trauma-hemorrhage.
05/2007
Downregulation of TLR4-dependent ATP production is critical for estrogen-mediated immunoprotection in Kupffer cells following trauma-hemorrhage.
05/2007
Downregulation of migration inhibitory factor is critical for estrogen-mediated attenuation of lung tissue damage following trauma-hemorrhage.
04/2007
G protein-coupled receptor 30-dependent protein kinase A pathway is critical in nongenomic effects of estrogen in attenuating liver injury after trauma-hemorrhage.
02/2007
Flutamide attenuates pro-inflammatory cytokine production and hepatic injury following trauma-hemorrhage via estrogen receptor-related pathway.
01/2007
Tissue compartment-specific role of estrogen receptor subtypes in immune cell cytokine production following trauma-hemorrhage.
01/2006
Flutamide restores cardiac function after trauma-hemorrhage via an estrogen-dependent pathway through upregulation of PGC-1.
12/2005
PGC-1 upregulation via estrogen receptors: a common mechanism of salutary effects of estrogen and flutamide on heart function after trauma-hemorrhage.
Articles by Research Topic
Disease
32
Wounds and Injuries (Trauma)
10/2008 - 07/2005
29
Hemorrhage
10/2008 - 08/2005
3
Sepsis (Septicemia)
09/2007 - 10/2005
2
Hemorrhagic Shock
01/2006 - 07/2005
1
Shock
01/2006
Drug/Important Bio-Agent (IBA)
14
Estrogens (Estrogen)
FDA Link
10/2008 - 12/2005
5
Flutamide (Eulexin)
FDA Link
Generic
02/2007 - 07/2005
5
Estrogen Receptor beta
IBA
09/2006 - 01/2006
3
Peroxidase (Myeloperoxidase)
IBA
09/2007 - 02/2006
3
Estrogen Receptor alpha
IBA
09/2007 - 08/2006
3
Androstenediol
IBA
08/2006 - 08/2005
3
Estrogen Receptors
IBA
05/2006 - 07/2005
2
Protein Kinases (Protein Kinase)
IBA
04/2008 - 06/2007
1
Glycogen
IBA
10/2008
1
Chemokine CCL3
IBA
08/2008
1
Gonadal Steroid Hormones (Sex Hormones)
IBA
09/2007
1
Adenosine Triphosphate (ATP)
IBA
05/2007
1
GTP-Binding Proteins (G-Protein)
IBA
04/2007
1
Cyclic AMP-Dependent Protein Kinases (cAMP-Dependent Protein Kinase)
IBA
04/2007
1
Chemokine CCL2 (Monocyte Chemoattractant Protein 1)
IBA
03/2007
1
p38 Mitogen-Activated Protein Kinases
IBA
03/2007
1
Heme Oxygenase-1
IBA
09/2006
1
Estradiol (Estrace)
FDA Link
Generic
08/2006
1
PPAR gamma
IBA
07/2006
1
Heat-Shock Proteins (Heat-Shock Protein)
IBA
01/2006
1
Cyclic AMP Response Element-Binding Protein (Cyclic AMP-Responsive DNA-Binding Protein)
IBA
10/2005
1
Protein Kinase C-alpha
IBA
10/2005
1
Peroxisome Proliferators
IBA
08/2005